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In the STZ-induced diabetes rat model, most of the islet cells are denatured and necrotic, nuclear fragmentation, islet size and quantity decrease, and some exocrine acinar epithelial vacuolar degeneration, thus increasing the blood glucose level and inducing excessive reactive oxygen species by hyperglycemia

For instance, P115 and GRASP65 trigger apoptosis via the ERK/p53/PUMA pathway or Fas-mediated signaling, respectively, whereas Bruce and GAAP decrease apoptosis via caspase-9, Smac and p53 inhibition, and Ca 2+ signaling downregulation [93]

Nat Commun 6:8130