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By contrast, efficient secretion of the target protein can result in the protein having to be purified away from a small number of secreted host proteins, especially if the host cells are grown under serum-free conditions [7]

It is likely that induction of ferroptosis in disease will occur through the erosion of ferroptosis defences that are ineffectually restored by disrupted or fatigued metabolic pathways

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Moreover, the vulnerability of the aging hippocampus to excitotoxicity is reinforced by the impairment of astrocytic glutamate uptake [21]