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and members of the Shen lab and Mootha lab, and colleagues at Yale School of Medicine and Yale West Campus for fruitful discussions and feedback

This suggests that the GSH production process and cellular excretion of GSSG are factors that would impact the level of [ROS] in diseased retinas after all rods have degenerated

Sirtuin3 alleviated influenza A virus-induced mitochondrial oxidative stress and inflammation in lung epithelial cells via regulating poly (ADP-ribose) polymerase 1 activity

Here, we show that sustained iron overload, but not acute exposure, leads to a state of ferroptotic stress where nerve cells remain viable but become hypersensitive to oxidative injury

2A during the early stage of infection (13 days post-infection), the basal OCR was reduced by approximately 2030%, indicating that early events of HIV infection impacted mitochondrial respiration without changing maximal respiration or spare capacity (Fig