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However, one limitation is that rodents possess UOX in their bodies, necessitating the inhibition of UOX activity to mimic human metabolic defects

By contrast, SFN treatment leads to a desensitization of the IP 3 /Ca 2+ entry which could be attributed to genomic actions

Both compounds target the pituitary gland to stimulate growth hormone release through receptor-level mechanisms, yet they differ in pathway, half-life, and selectivity, producing distinct research profiles relevant to body composition, fat loss, and lean muscle outcomes

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Methods GHSR expression and purification All primers used in this study are listed in Supplementary Table 2