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Such neuronal death in a caspase-independent manner was described in stroke, in which NG2 glia produce Bcl-2/E1B-19K-interacting protein 3 (BNIP3), a proapoptotic member of the Bcl-2 family proteins (Li et al., 2013)

When ROS accumulates excessively, it will cause oxidative stress damage to cells

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These conditions can be primary (idiopathic) or secondary to other neurological disorders, such as Parkinson's disease, and are typically managed through physical therapy, medications like anticholinergics, or botulinum toxin injections

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