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Mitochondrial dysfunction contributes to oncogene-induced senescence

2020;133:jcs247353

By jointly targeting multiple cell death pathways such as autophagy, apoptosis, pyroptosis, necroptosis, and ferroptosis, it is expected to significantly improve the treatment effect of CD and overcome drug resistance through the synergistic regulation of intestinal inflammation, barrier function, and immune response

Dev Period Med

Consistent with this, analysis of multiple TCGA datasets correlating p53 protein (RPPA) and SLC7A11 mRNA (RNAseq) expression demonstrated the same relationship (Supplementary Fig