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This combination of simultaneous lean-mass and fat-mass change is one of the more frequently cited findings distinguishing IGF-1 LR3 research from single-pathway compounds

J Alzheimers Dis 9: 101115, 2006

Pepe M, Longton G, Janes H

This mechanism involves: Sequestration of monomeric actin (G-actin) to prevent premature polymerization Promotion of actin polymerization into functional microfilaments (F-actin) when needed Enhanced cellular migration through cytoskeletal reorganization Facilitation of cell shape changes necessary for wound healing Research indicates TB-500 may work with the Arp2/3 protein complex to orchestrate actin polymerization, though this mechanism requires further clarification[4]