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Relative contributions of thioltransferase-and thioredoxin-dependent systems in reduction of low-molecular-mass and protein disulphides

Future efforts should focus on the development of brain-penetrant inhibitors, rational combination therapies, and predictive biomarkers to facilitate clinical translation

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Administering UA to adult rats either 24 h before middle cerebral artery occlusion (62.5 mg/kg, intraperitoneally) or 1 h after reperfusion (16 mg/kg, intravenously) can protect cultured hippocampal neurons from cell death induced by brain ischemia-related damage, including exposure to excitatory amino acids, glutamate, and the metabolic poison cyanide [139]