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, Expression of Abcc1 , which encodes MRP1 that is responsible for extracellular glutathione efflux (Figure 1E), also increased after IRI (Figure 3M)

In vivo pharmacokinetic evaluation showed that these SLNs increased oral bioavailability by 4.3-fold compared with the marketed formulation, highlighting their ability to overcome poor solubility and first-pass metabolism [83]

Dara L, Johnson H, Suda J et al (2015) Receptor interacting protein kinase 1 mediates murine acetaminophen toxicity independent of the necrosome and not through necroptosis

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This suggests that the platform may also help influence oxidative stress and tumour-related inflammation, although further studies are needed to better understand these effects