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doi: 10.1007/978-94-007-4549-0_28 39 HeinolainenKKaramanSD'AmicoGTammelaTSormunenREklundLet al

Furthermore, advancements in mRNA modification, Such as 3-end extensions, provide additional tools to enhance therapeutic efficacy by optimizing stability and translational capacity

However, CoA does not fit well within the modeled catalytic pocket, consistent with our experimental findings that ABHD11 does not hydrolyze the glutaryl-CoA thioester

7 , e2474 (2016)

Animal models and clinical studies of ASD suggest that abnormalities in GABAergic neurons, signaling pathways, and related genes may play an important role in the pathogenesis of ASD, leading to abnormal levels of GABA in the blood and brain tissue of individuals with ASD