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Interestingly, short-term incubation with FLT3i strongly depleted C/EBP protein expression without modification of its inhibitory phosphorylation at Ser21 (ratio P S21 -C/EBP / total C/EBP p42

To assess whether the HIF-dependent potentiation of cell death is due to synergizing with compounds that reduce GSH pools, we used buthionine sulfoximine (BSO), which inhibits de novo GSH biosynthesis

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HGF needs to form homodimers and/or multimers, prior to its activation of Met (Chirgadze et al., 1999