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Overall, glutathione depletion, oxidative stress, and mitochondrial damage are the critical underlying mechanisms of ALI ( Figure 1 )

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As a result, current dietary Cd exposure limits, which are computed by adjusting the excretion rates of Cd and biomarkers of kidney effects to E cr , underestimate the severity of Cd-induced nephrotoxicity

Sequential additions of 0.1% trifluoroacetic acid (TFA) in acetonitrile and 0.1% TFA in 50% acetonitrile were added to the S-trap, centrifuged and pooled