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Expression of methylthioadenosine phosphorylase cDNA in p16-, MTAP- malignant cells: restoration of methylthioadenosine phosphorylase-dependent salvage pathways and alterations of sensitivity to inhibitors of purine de novo synthesis

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Taken together, the prioritized Glu/Gln pathway in IPH+ plaque seems to implicate shifts in macrophage metabolism

40 Therefore, the interaction between antioxidants and iNOS/COX-2 presents a possible target for inflammation management