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& DeFea, K

Osborn, M

Preclinical research associates it with new capillary growth, nitric oxide signaling, and the FAK-paxillin pathway involved in repair-cell attachment and migration. These mechanisms provide the rationale for its use as the perfusion component of the stack

The microsphere-released drug was effectively taken up by A549 cells and had a strong inhibitory effect on cell migration and invasion by inducing apoptosis and cell cycle arrest in the G2/M phase (Xiong et al., 2021)