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Fe 2+ absorbed into the blood is oxidized to Fe 3+ by ceruloplasmin, bound by transferrin, and then transported to tissues

Another study suggests that the accumulation of 2-HG is likely due to increased transamination activity mediated by glutamate Oxaloacetate Transaminase(GOT1), resulting in promoter methylation at the Foxp3 locus and reduced TcB induction (139)

The implication of ferroptosis in IBD pathology, as outlined above, is illustrated in Fig