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Complex I dysfunction results in substantially reduced ATP production, creating an energy deficit that directly compromises sperm motility and cellular function ( 3.4 Molecular pathway interaction networks The reproductive toxicity induced by EEDs results from complex crosstalk among oxidative stress responses, metabolic disruption, and immune system activation rather than isolated pathway effects

This study adds to the growing research on how antioxidants like GSH control infection

doi: 10.1111/j.0105-2896.2004.00180.x 50 ShapiraSSpeirsKGersteinACaamanoJHunterCA

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Sigma, Taufkirchen, Germany) solution (15 g/ml FDA in 1 DPBS)