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Since previous studies have linked the activation of NRF2 with the repression of lipogenic gene expression 64 , we reasoned that GSH depletion causes oxidation and consequent inactivation of KEAP1 (a repressor of NRF2), thus leading to increased antioxidant and decreased lipogenic gene expression

However, these processes are disrupted when the autoimmune cascade is triggered and the thyroid is attacked

The toxicity of A peptides proteinopathy affects immediately the whole myelin-axon, which forms a couple with multiple functional and metabolic relationships [181]

Mold toxicity is a hidden epidemic, silently affecting countless individuals who struggle with unexplained symptoms

Heinz bodies can lead to hemolysis, potentially resulting in anemia