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Whereas preclinical and clinical research have extensively expounded on these target organ interventions, clinical application and instance-based evidence on clinical translatability are needed

10.1016/j.talanta.2010.02.025 22 ChaneyA.WilliamsS

has elegantly demonstrated that NRF2 is a key suppressor of ILC2 activation, our study provides the missing upstream link, identifying melatonin, a canonical circadian hormone, as a critical activator of the NRF2-Glutathione axis in ILC2s (64)