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Furthermore, pharmacologically induced PIEZO1 activation (through Yoda1), combined with mechanical stretch, upregulates Lox expression in tendon fascicle explants

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Patients who are truly deficient receive the treatment they need including injections when clinically warranted and patients who are not deficient are guided toward more appropriate solutions

Myocardial remodelling characterized by fibrosis and hypertrophy, impaired microcirculation, abnormal protein function caused by advanced glycation end products, changes in myocardial metabolism and abnormal mitochondrial function have been noted as associated factors of diabetic cardiomyopathy [14]

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