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N-Acetyl cysteine inhibits cell proliferation and differentiation of LPSInduced mc3t3-E1 cells via regulating inflammatory cytokines

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In mouse models, the team introduced combined therapy including PD-L1 blocker and cyst(e) inase (a bioengineered enzyme that degrades cystine and cysteine) and such treatment can enhance the efficacy of CTL-mediated anticancer effects, supporting the idea that inducing ferroptosis could be considered a productive way for treating cancer and overcoming immunotherapy resistance (Wang et al., 2019)

Collectively, GGCT functions as a metabolic rheostat that orchestrates mitochondrial homeostasis through diverting Gln flux toward glutathione biosynthesis to fortify redox defense, while enhancing Glc-fueled TCA cycle anaplerosis to sustain cell proliferation

p53 can downregulate the expression of the Xc system component SLC7A11, inhibiting cystine intake and inducing ferroptosis [61]