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Glucagon receptor activation is more complicated: glucagon can raise glucose, but in engineered multi-agonists it may also increase energy expenditure, fat oxidation, and liver-metabolic effects when balanced against GLP-1 activity

& Ulfendahl, M

In CIRS, inappropriate suppression of ADH relative to elevated osmolality suggests hypothalamic injury or receptor resistance, contributing to polydipsia, frequent urination, orthostatic intolerance, and salt wasting