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Theat least partial [24,97]structural and chemical similarity to physiological bio-molecules can explain the ability of thiol-bound Mercury(II) species to cross biological membranes by using the physiological transport systems of the natural substrates [21] and its ability to undergo chemical modifications operated by the enzymes which process bio-molecules such as those of the soluble thiolome

The primary victims of this relentless oxidative stress are the mitochondria, the microscopic powerhouses responsible for generating ATP

Figure 16 shows key design considerations for redox-responsive drug delivery systems

Rheumatology 54 (2), 363368