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Therefore, we hypothesize that BPC157 may reduce intracellular ROS levels through an antioxidant mechanism similar to that reported in J Clin Invest [19], thereby stabilizing BACH1 protein and enabling its pro-angiogenic function

Mutations in the Casitas B-cell lymphoma (Cbl)-binding domain are demonstrated to be oncogenic because binding of Cbl to Y1003 causes Met ubiquitination that is vital to maintenance of physiological Met activation and prevention of continued activation of Met (Peschard and Park, 2007)

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In combination trials with other agents, BPC-157 appears to facilitate overall musculoskeletal healing when used in conjunction with mechanical loading or other regenerative agents