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Thus, although some of the damage in this experimental model of colitis reflects a direct erosive effect of TNBS and ethanol, their combined administration to rats reflects the in vivo operation of the damaging mechanisms elicited by TNBS, namely GSH depletion and ROS generation, indicating that ethanol facilitates the access of TNBS to the cell interior ensuring TNBS-induced GSH depletion and ROS generation

These results suggest that UgsL accelerates the degradation of OsSRF3 by enhancing its association with the ubiquitin-26S proteasome

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Simmilarly, in ErbB2 + breast cancer models, Nandi et al

Over the following 30 years, more than 130 peer-reviewed papers have followed, the vast majority from his group and primarily in animal models