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Many cardiovascular risk factors such as diabetes mellitus, hypertension and hyperlipidemia are demonstrated to stimulate the interaction of Cav-1 and eNOS, reduce nitric oxide production resulting in endothelial dysfunction and subsequently the formation of atherosclerotic lesion 4143

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Over time, this cumulative strain may lead to ligament laxity

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At the same time, edema almost completely vanished in the BPC 157 rats (Figure 20)