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10.1002/smll.201905233 Small 111 RimassaL.AssenatE.Peck-RadosavljevicM.PrachtM.ZagonelV.MathurinP.et al (2018)

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BDNF and TrkB Upregulation The most extensively documented mechanism of Semax in pre-clinical research is its rapid upregulation of brain-derived neurotrophic factor (BDNF) and its primary signalling receptor, tropomyosin receptor kinase B (TrkB)