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Description

Rinella M, Charlton M

DOI:10.1016/S0278-5846(96)00103-0 [ Links ] 37

Country of Origin:USA Manufacturer/Importer/Packed By:Toniiq

The above conclusions were also confirmed in FER-1-treated OA model mice (48)

Therefore, the major oxidative burst during I/R occurs after the reexposure to oxygen during the so called warm ischemia time.7 Although the molecular mechanisms involved in I/R cellular responses are still not fully understood, a major role seems to be played by APE/Ref-1 and NF-B proteins in liver.42 Overexpression of APE/Ref-1 by adenoviral vector resulted in suppression of the reperfusion-generated oxidative stress in a mouse model.42 Liver transplantation (LT) is at present the only available choice for the treatment of end-stage hepatic disease